Inhibitors of the electron transport chain exert their effects through a variety of mechanisms.

a. rotenone rotenone atractyloside azide blocks electron transport and proton pumping via inhibition of complex iv.
b. azide rotenone atractyloside blocks electron transport and proton pumping via inhibition of complex i.
c. blocks electron transport and atp synthesis by inhibiting the exchange of atp and adp across the inner mitochondrial membrane.
d. blocks atp synthesis without inhibiting electron transport by dissipating the proton gradient.

The mechanism of inhibition of various electron transport inhibitors are given below.

Explanation:

a Rotenone  rotenone blocks the electron transport and proton pumping via inhibition of complex 1.

b Azide  it blocks electron transport and proton pumping via inhibition of complex 4 or Cytochrome A oxidase.

c Atractyloside  it blocks electron transport and ATP synthesis by inhibiting the exchange of ATP and ADP across the inner mitochondrial membrane.

answer; atp and nadph;

"1. it should be, it has been for about 20 years, and it's really not much different in the end than breeding animals to have particular traits. there are always concerns about genetically engineered animals/plants affecting the environment in unexpected negative ways, but i can't think of an example where the fears didn't turn out to be baseless.  

first, as you point out, it would be a great advance in pharmacology, to be able to tailor drugs and their doses to me. the pros involve better medical treatment. the cons involve privacy issues like insurance companies denying coverage to people with genetic risk factors. looking at dna markers wouldn't affect the frequency of genetic diseases, but it would allow detection of them earlier. and ok, actually i suppose that if people used this information to avoid having children with genetic diseases, some might decrease in frequency."