You study color variants of Arabidopsis hypotheticus, a plant with red flowers. You have obtained three pure-breeding mutant lines, all named for their place of origin. Two lines have white flowers (Aberdeen White and Victoria White), and one has yellow flowers (Kansas Yellow).
You begin your analysis by crossing each line with wild-type plants and selfing the F1 to produce an F2 generation.[Crosses between Aberdeen White and wild type, Victoria White and wild type, and Kansas Yellow and wild type] From these crosses you conclude that the mutations in all three lines are recessive to the wild type.

Crossing lines with the same recessive phenotype
You continue your analysis by crossing the Aberdeen White and Victoria White lines. This time you count the actual numbers of progeny in the two F2 phenotypic classes.
[Cross between Aberdeen White and Victoria White]
You also self several wild-type plants from the F2 and determine that some of them are pure-breeding. What can you conclude from these results?

From the results we may conclude that;

1. Aberdeen White and Kansas Yellow are alleles of different loci.

2. The lack of wild-type progeny in the F2 is the result of independent assortment

3. The F2 ratio is approximately 9:7

4. The F2 ratio results from complementary gene interactionction.

Explanation:

The tables below is a representation of the question with appropriate labelling.

In genetics a loci is a fixed position on a chromosome that may be occupied by one or more genes.

Wild type (WT) flower describes the phenotype of the typical form of a species as it occurs in nature.

A progeny — is a offspring or descendants.

The Laws of independent assortment describes that alleles for one trait segregate independently from alleles for another trait in metaphase of meiosis. Each allele combination is equally likely to occur in the progeny.

Like we have in the question the experiment showed that the two genes interact to produce the overall flower color (the phenotype); when genes work in tandem to produce a single gene product, it is described as a complementary gene interaction.

The wild-type action from both genes is required to produce the wild-type phenotype.

Mutation of one or both genes produce a mutant phenotype.

the answer to your question is tooth decay.