With age, somatic cells are thought to accumulate genomic "scars" as a result of the inaccurate repair of double-strand breaks by nonhomologous end-joining (NHEI). Estimates based on the frequency of breaks in primary human fibroblasts suggest that by age 70 each human somatic cell may carry some 2000 NHEl-induced mutations due to inaccurate repair. If these mutations were distributed randomly around the genome, how many genes would you expect to be affected? Would you expect cell function to be compromised? Why or why not? (Assume that 2% of the genome-l.5% coding and 0.5% regulatory-is crucial information.)

Agroup of students is studying convection currents. they fill two identical balloons with the same amount of helium. one balloon is placed in a freezer and the other in an area with warm air. after 10 minutes, the balloons are released from a height of 1 meter. which of the following do the students most likely observe? question 8 options: the cold balloon expands and rises. the warm balloon shrinks and sinks. the balloons rise at the same rate. both balloons are the same size. the ballons both rise. the cold ballon is larger than the warm balloon. the warm balloon expands and rises. the cold balloon shrinks and sinks.