Why is it essential for SARS-CoV2 RNA be translated on ribosomes before it can be replicated?

The SARS-CoV-2 non-structural protein 1 (Nsp1), also referred to as the host shutoff factor, suppresses host innate immune functions. By combining cryo-electron microscopy and biochemistry, we show that SARS-CoV-2 Nsp1 binds to the human 40S subunit in ribosomal complexes, including the 43S pre-initiation complex and the non-translating 80S ribosome. The protein inserts its C-terminal domain into the mRNA channel, where it interferes with mRNA binding. We observe translation inhibition in the presence of Nsp1 in an in vitro translation system and in human cells.

i believe it would be c: tons.

sorry to break it to ya but no one can answer your question it's just

jk but i am serious about the part that most people cant answer it beacuse you did not mention the section you are talking about. but it would if you added some detail and were a bit more specific.